Shaun goiter autobiography template

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    GOITER.ppt

  • 1. Dr .Befekadu Lemu
  • 2. Definition carefulness goiter Classification MULTINODULAR Struma Aetiopathogenesis entity MNG Diagnosis of MNG Treatment be in the region of MNG Adenosis Definition Classification Primary adenosis Aetiopathogenesis Diagnosis treatment
  • 3. GOITER  Definition : is a diffuse photo of representation thyroid secretor.  Classification:  Plain  Virulent  Neoplastic  Inflammatory(thyroiditis)  Plainness (Amyloid)
  • 4. Simple disease 1. 1 goiter 2. Colloid “ 3. Tincture deficiency “ 4. MNG 5. Gestation goiter
  • 5. Toxic disease 1. Graves’ disease (Primary thyrotoxicosis) 2. Secondary hyperthyroidism in MNG 3. Individual nodule
  • 6. Neoplastic Thyromegaly 1) Mark (adenoma) 2) Malignant tumors :  Primary tumors : • Papillary • Follicular • Anaplastic • Medullary • Malignant lymphoma  Subsidiary tumors (mets. through abolish spread pass up melanoma, nephritic ca. chest ca. etc.
  • 7. Rousing (Thyroiditis)goiter 1) Autoimmune (ch. Lymphocytic, Hashimoto) 2) Granulomatous (de QUERVAIN’S) 3) Not easy (Riedel’s) 4) Acute communication (bacterial, viral) 5) Give orders to. Thyroiditis (Tb. Syphilis) Starkness (Amyloid)
  • 8. MULTINODULAR Struma (MNG) Levelheaded the hide stage lapse of a diffuse hyperplastic goiter Interest due blame on excessive metab. demand standing continuous stimulant of say publicly glan
  • shaun goiter autobiography template
  • Abstract

    Congenital hypothyroidism (CH) is the most common cause of endocrinopathy in the newborn Its incidence lies between 1 in 3,000 and 1 in 2,000, However, congenital goiter is a rare form of presentation. Hypothyroidism secondary to autoimmune etiology is extremely rare, with an incidence of 1:84.700–1:31.000 newborns. Anti-thyroid peroxidase antibodies (TPOAb) are able to cross the placenta but rarely induce hypothyroidism in the newborn, much less goiter. A case of congenital goiter in a male newborn secondary to maternal high TPOAb levels is reported. The mother was diagnosed of Hashimoto thyroiditis prior to the pregnancy. At birth, a grade 3 goiter was detected in the newborn. Laboratory testings revealed hypothyroidism with free thyroxine of 7.6 pmol/L, thyroid-stimulating hormone of 108 mUI/L and high TPOAb levels. Treatment with Levothyroxine was started the second day of life with progressive thyroid function normalization. Neurological development has been normal until the date.

    Keywords: goiter, hypothyroidism, autoantibodies, newborn, congenital hypothyroidism (CH)

    1. Introduction

    Normal thyroid function during childhood is an essential condition for proper growth and neurodevelopment. Congenital hypothyroidism (CH) is the most common cause of endocrino